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In this Perspective, we describe novel molecular underpinnings and metabolic drivers of ferroptosis, analyse the functions and mechanisms of ferroptosis in tumours, and highlight prostate cancer-specific susceptibilities to ferroptosis by connecting ferroptosis pathways to the distinctive metabolic reprogramming of prostate cancer cells
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By addressing the complex interplay of forces and molecular processes that contribute to ACL injuries, it is possible to improve outcomes for individuals at risk of or recovering from these debilitating injuries (Table 2)
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