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Since HGF may be a powerful enhancer of IDO1 [31] , it is plausible that increased concentrations of HGF, released in either an autocrine or paracrine way, can induce IDO1 upregulation
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While these experimental observations outline a plausible network connecting iron dysregulation, tau pathology, and ferroptosis, it is essential to distinguish between mechanistic associations established in model systems and causative evidence in human disease
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