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Allosteric inhibition of tRNA synthetase Gln4 by N-pyrimidinyl-β-thiophenylacrylamides exerts highly selective antifungal activity: Cell Chemical Biology
Finally, we obtained 18 proteins
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Thus, similar to the observed protective functions of IgG4 antibodies, MuSK autoantibodies exert their pathogenicity through blocking the interaction between MuSK and LRP4 that is required for the clustering of the AChR (83, 84)
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